Gastrointestinal (GI): NSAIDs that block COX-one activity have correlations with GI complications due to inhibition from the synthesis of gastroprotective agents which include PGE2 along with other prostaglandins. This inhibition results in decreased mucin generation by gastric epithelial cells, considerably less bicarbonate secretion, and less epithelial mobile turnover, between other https://chestert973ikj2.snack-blog.com/profile